Journal article icon

Journal article

Magnetic resonance imaging of the regenerating neonatal mouse heart

Abstract:
After myocardial infarction (MI) the human heart is unable to regenerate lost tissue, leading to scarring, pathological remodeling and progression to heart failure. The study of animal models that can intrinsically regenerate their heart, therefore, offers therapeutic insight into targeting tissue restoration. In 2011, the first evidence of mammalian heart regeneration was reported by Hesham Sadek, Eric Olson and colleagues1. Following surgical resection of ~15% of the left ventricle apex of a oneday old (P1) neonatal mouse, the heart fully regenerated by 21-days post-injury, whereas if the procedure was repeated one week later on a post-natal day 7 (P7) mouse heart, fibrosis and scarring ensued, recapitulating the adult wound-healing response. The mechanism of regeneration observed involved proliferation of resident cardiomyocytes, analogous to that described in the adult zebrafish heart2. Since the original study others have described neonatal myocardial regeneration after alternative insults, such as MI3. However, there is also controversy surrounding the extent of heart regeneration during the first weeks of life, whereby it was reported regeneration did not occur in the P1 heart and was replaced by long-term fibrosis (180 days) with extensive cardiac remodelling4.
Publication status:
Published
Peer review status:
Peer reviewed

Actions


Access Document


Files:
Publisher copy:
10.1161/CIRCULATIONAHA.118.036086

Authors


More by this author
Institution:
University of Oxford
Division:
Medical Sciences Division
Department:
Physiology Anatomy and Genetics
Role:
Author
More by this author
Institution:
University of Oxford
Division:
Medical Sciences Division
Department:
Physiology Anatomy and Genetics
Role:
Author
More by this author
Institution:
University of Oxford
Division:
Medical Sciences Division
Department:
Oncology
Role:
Author
More by this author
Institution:
University of Oxford
Division:
Medical Sciences Division
Department:
Oncology
Role:
Author
More by this author
Institution:
University of Oxford
Division:
Medical Sciences Division
Department:
RDM
Sub department:
RDM Cardiovascular Medicine
Role:
Author


More from this funder
Funding agency for:
Masters, M
Grant:
289600
More from this funder
Funding agency for:
Gunadasa-Rohling, M
Schneider, J
Riley, P
Grant:
RM/13/3/30159
RM/13/3/30159
RM/13/3/30159


Publisher:
American Heart Association
Journal:
Circulation More from this journal
Volume:
138
Issue:
21
Pages:
2439–2441
Publication date:
2018-11-19
Acceptance date:
2018-08-27
DOI:
EISSN:
1524-4539
ISSN:
0009-7322


Pubs id:
pubs:926045
UUID:
uuid:dc97e670-3ef2-469f-8600-f5e0d7154a3d
Local pid:
pubs:926045
Source identifiers:
926045
Deposit date:
2018-10-10

Terms of use



Views and Downloads






If you are the owner of this record, you can report an update to it here: Report update to this record

TO TOP