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Thesis

The cell-intrinsic and microenvironmental basis of impaired B lymphopoiesis in Trisomy 21 fetal bone marrow

Abstract:
Background: Children with Down syndrome, which is caused by trisomy of chromosome 21 (T21), have an increased risk of developing B cell acute lymphoblastic leukaemia (ALL) and an increased frequency and severity of infections. Individuals with Down syndrome also have B lymphopenia and an increased frequency of autoimmune diseases often in association with circulating self-reactive antibodies. Our lab has previously shown this striking reduction in B cell frequency can be trac... Expand abstract

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Authors

More by this author
Institution:
University of Oxford
Division:
MSD
Department:
Paediatrics
Role:
Author
ORCID:
https://orcid.org/0009-0003-9639-5763

Contributors

Role:
Contributor
Institution:
University of Oxford
Division:
MSD
Department:
Radcliffe Department of Medicine
Role:
Contributor
ORCID:
No
Role:
Contributor
ORCID:
0009-0007-1902-4474
Institution:
University of Oxford
Division:
MSD
Department:
Paediatrics
Role:
Supervisor
Institution:
University of Oxford
Division:
MSD
Department:
Paediatrics
Role:
Supervisor
ORCID:
0000-0002-6094-6397


More from this funder
Funder identifier:
https://ror.org/054225q67


DOI:
Type of award:
DPhil
Level of award:
Doctoral
Awarding institution:
University of Oxford

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