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Prostaglandin D2 activates group 2 innate lymphoid cells through chemoattractant receptor-homologous molecule expressed on TH2 cells

Abstract:

Background Activation of the group 2 innate lymphoid cell (ILC2) population leads to production of the classical type 2 cytokines, thus promoting type 2 immunity. Chemoattractant receptor-homologous molecule expressed on TH2 cells (CRTH2), a receptor for prostaglandin D2 (PGD2), is expressed by human ILC2s. However, the function of CRTH2 in these cells is unclear.

Objectives We sought to determine the role of PGD2 and CRTH2 in human ILC2s and compare it with that of the established ILC2 activators IL-25 and IL-33.

Methods The effects of PGD2, IL-25, and IL-33 on the cell migration, cytokine production, gene regulation, and receptor expression of ILC2s were measured with chemotaxis, ELISA, Luminex, flow cytometry, quantitative RT-PCR, and QuantiGene assays. The effects of PGD2 under physiologic conditions were evaluated by using the supernatant from activated mast cells.

Results PGD2 binding to CRTH2 induced ILC2 migration and production of type 2 cytokines and many other cytokines. ILC2 activation through CRTH2 also upregulated the expression of IL-33 and IL-25 receptor subunits (ST2 and IL-17RA). The effects of PGD2 on ILC2s could be mimicked by the supernatant from activated human mast cells and inhibited by a CRTH2 antagonist.

Conclusions PGD2 is an important and potent activator of ILC2s through CRTH2 mediating strong proallergic inflammatory responses. Through IgE-mediated mast cell degranulation, these innate cells can also contribute to adaptive type 2 immunity; thus CRTH2 bridges the innate and adaptive pathways in human ILC2s.

Publication status:
Published
Peer review status:
Peer reviewed

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Publisher copy:
10.1016/j.jaci.2013.10.056

Authors

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Institution:
University of Oxford
Division:
MSD
Department:
NDM
Sub department:
NDM Experimental Medicine
Role:
Author
More by this author
Institution:
University of Oxford
Division:
MSD
Department:
RDM
Role:
Author
More by this author
Institution:
University of Oxford
Division:
MSD
Department:
NDM
Sub department:
NDM Experimental Medicine
Role:
Author


More from this funder
Funder identifier:
https://ror.org/03x94j517
Funding agency for:
Ogg, G
More from this funder
Funder identifier:
https://ror.org/029chgv08
Funding agency for:
Klenerman, P
More from this funder
Funder identifier:
https://ror.org/02caz1f24
Funding agency for:
Klenerman, P
Xue, L
Ogg, G
More from this funder
Funder identifier:
https://ror.org/0187kwz08
Funding agency for:
Ogg, G
More from this funder
Funding agency for:
Klenerman, P


Publisher:
Elsevier
Journal:
Journal of Allergy and Clinical Immunology More from this journal
Volume:
133
Issue:
4
Pages:
1184-1194.e7
Publication date:
2013-12-31
Acceptance date:
2013-10-28
DOI:
EISSN:
1097-6825
ISSN:
0091-6749


Language:
English
Keywords:
Pubs id:
pubs:462662
UUID:
uuid:5882c732-0862-474b-845c-b0f41bf292ba
Local pid:
pubs:462662
Source identifiers:
462662
Deposit date:
2014-07-11
ARK identifier:

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