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Empathy is not in our genes

Abstract:
In academic and public life empathy is seen as a fundamental force of morality – a psychological phenomenon, rooted in biology, with profound effects in law, policy, and international relations. But the roots of empathy are not as firm as we like to think. The matching mechanism that distinguishes empathy from compassion, envy, schadenfreude, and sadism is a product of learning. Here I present a dual system model that distinguishes Empathy1, an automatic process that catches the feelings of others, from Empathy2, controlled processes that interpret those feelings. Research with animals, infants, adults and robots suggests that the mechanism of Empathy1, emotional contagion, is constructed in the course of development through social interaction. Learned Matching implies that empathy is both agile and fragile. It can be enhanced and redirected by novel experience, and broken by social change.
Publication status:
Published
Peer review status:
Peer reviewed

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Publisher copy:
10.1016/j.neubiorev.2018.11.001

Authors

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Institution:
University of Oxford
Division:
MSD
Department:
Experimental Psychology
Oxford college:
All Souls College
Role:
Author


Publisher:
Elsevier
Journal:
Neuroscience and Biobehavioral Reviews More from this journal
Volume:
95
Pages:
499-507
Publication date:
2018-11-03
Acceptance date:
2018-11-02
DOI:
ISSN:
0149-7634


Keywords:
Pubs id:
pubs:938390
UUID:
uuid:ff6deb65-f828-4ab7-a663-e149f4269d1e
Local pid:
pubs:938390
Source identifiers:
938390
Deposit date:
2018-11-06
ARK identifier:

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