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Population-specific causal disease effect sizes in functionally important regions impacted by selection

Abstract:
Many diseases exhibit population-specific causal effect sizes with trans-ethnic genetic correlations significantly less than 1, limiting trans-ethnic polygenic risk prediction. We develop a new method, S-LDXR, for stratifying squared trans-ethnic genetic correlation across genomic annotations, and apply S-LDXR to genome-wide summary statistics for 31 diseases and complex traits in East Asians (average N = 90K) and Europeans (average N = 267K) with an average trans-ethnic genetic correlation of 0.85. We determine that squared trans-ethnic genetic correlation is 0.82× (s.e. 0.01) depleted in the top quintile of background selection statistic, implying more population-specific causal effect sizes. Accordingly, causal effect sizes are more population-specific in functionally important regions, including conserved and regulatory regions. In regions surrounding specifically expressed genes, causal effect sizes are most population-specific for skin and immune genes, and least population-specific for brain genes. Our results could potentially be explained by stronger gene-environment interaction at loci impacted by selection, particularly positive selection.
Publication status:
Published
Peer review status:
Peer reviewed

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Publisher copy:
10.1038/s41467-021-21286-1

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Funder identifier:
https://ror.org/05h1kgg64
Grant:
R01 HG006399
U01 HG009379
R37 MH107649
R01 MH101244
R01 CA222147


Publisher:
Nature Research
Journal:
Nature Communications More from this journal
Volume:
12
Issue:
1
Article number:
1098
Place of publication:
England
Publication date:
2021-02-17
Acceptance date:
2021-01-15
DOI:
EISSN:
2041-1723
Pmid:
33597505


Language:
English
Pubs id:
1280089
Local pid:
pubs:1280089
Deposit date:
2025-08-14
ARK identifier:

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