Conference item icon

Conference item : Abstract

Fibroblast growth factor 2 promotes regeneration of cartilage by attracting mesenchymal stem cells to the site of cartilage injury

Abstract:

An important response of cartilage to injury is release of fibroblast growth factor 2 (FGF2) from the pericellular matrix. Deletion of FGF2 in mice, leads to worsening cartilage degradation both spontaneously and after surgical induction of osteoarthritis (OA); thereby indicating a chondroprotective role for FGF2. One mechanism by which FGF2 could protect the joint is by promoting intrinsic repair after injury.


Healing of focal cartilage defects in mice has been shown to be dependent upon background strain and age, with young DBA1 mice having superior regenerative capacity. It is presumed that repair is dependent upon mesenchymal stem cell (MSC) migration to the injured site, followed by the process of chondrogenesis. Here we test whether FGF2 is involved in these processes.

Publication status:
Published
Peer review status:
Reviewed (other)

Actions


Access Document


Files:
Publisher copy:
10.1016/j.joca.2018.02.090

Authors


More by this author
Institution:
University of Oxford
Division:
Medical Sciences Division
Department:
NDORMS
Role:
Author
More by this author
Institution:
University of Oxford
Division:
Medical Sciences Division
Department:
NDORMS; KIR
Role:
Author
More by this author
Institution:
University of Oxford
Division:
Medical Sciences Division
Department:
NDORMS; KIR
Role:
Author


Publisher:
Elsevier
Host title:
Osteoarthritis and Cartilage
Journal:
World Congress on Osteoarthritis (OARSI 2018) More from this journal
Volume:
26
Issue:
S1
Pages:
S37
Publication date:
2018-04-29
Acceptance date:
2018-01-08
DOI:


Subtype:
Abstract
Pubs id:
pubs:856232
UUID:
uuid:dbdb76c5-6d92-4038-b35e-ab854b9fe7e7
Local pid:
pubs:856232
Source identifiers:
856232
Deposit date:
2018-06-13

Terms of use



Views and Downloads






If you are the owner of this record, you can report an update to it here: Report update to this record

TO TOP