- Abstract:
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Introduction: Diseased tendons are characterised by fibrotic scar tissue, which adversely affects tendon structure and function and increases the likelihood of reinjury. The mechanisms and expression profiles of fibrosis in diseased tendon is understudied compared to pulmonary and renal tissues, where TGFβ and its associated superfamily are known to be key drivers of fibrosis and modulate extracellular matrix homeostasis. We hypothesised that differential expression of TGFβ superfamily member...
Expand abstract - Publication status:
- In press
- Peer review status:
- Peer reviewed
- Version:
- Accepted manuscript
- Files:
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(pdf, 277.2KB)
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- Publisher:
- BioMed Central Publisher's website
- Journal:
- Arthritis Research and Therapy Journal website
- Publication date:
- 2016-01-01
- EISSN:
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1478-6362
- ISSN:
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1478-6354
- URN:
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uuid:cf87477d-99c4-48e7-b8d0-5106604419ee
- Source identifiers:
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598588
- Local pid:
- pubs:598588
- Keywords:
- Copyright holder:
- Goodier et al.
- Copyright date:
- 2016
- Notes:
- © 2016 Goodier et al. Published by BioMed Central. Open Access: This article is distributed under the terms of the Creative Commons Attribution 4.0 International License. This is the accepted manuscript version of the article. The final version will be available online from BioMed Central after publication.
Journal article
Comparison of TGFβ expression in healthy and diseased human tendon
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Funding
NIHR Oxford Musculoskeletal Biomedical Research Unit
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Girdlestone Scholarship
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