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Comparison of TGFβ expression in healthy and diseased human tendon

Abstract:

Introduction: Diseased tendons are characterised by fibrotic scar tissue, which adversely affects tendon structure and function and increases the likelihood of reinjury. The mechanisms and expression profiles of fibrosis in diseased tendon is understudied compared to pulmonary and renal tissues, where TGFβ and its associated superfamily are known to be key drivers of fibrosis and modulate extracellular matrix homeostasis. We hypothesised that differential expression of TGFβ superfamily member...

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Publication status:
In press
Peer review status:
Peer reviewed

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Institution:
University of Oxford
Division:
MSD
Department:
NDORMS
Role:
Author
More by this author
Institution:
University of Oxford
Division:
MSD
Department:
NDORMS
Role:
Author
More by this author
Institution:
University of Oxford
Division:
MSD
Department:
NDORMS
Role:
Author
More by this author
Institution:
University of Oxford
Division:
MSD
Department:
NDORMS
Role:
Author
More by this author
Institution:
University of Oxford
Division:
MSD
Department:
NDORMS
Role:
Author
More from this funder
Name:
Arthritis Research UK
Funding agency for:
Dakin, S
Snelling, S
Grant:
20506
20087
More from this funder
Name:
NIHR Oxford Musculoskeletal Biomedical Research Unit
Publisher:
BioMed Central
Journal:
Arthritis Research and Therapy More from this journal
Publication date:
2016-01-01
EISSN:
1478-6362
ISSN:
1478-6354
Keywords:
Pubs id:
pubs:598588
UUID:
uuid:cf87477d-99c4-48e7-b8d0-5106604419ee
Local pid:
pubs:598588
Source identifiers:
598588
Deposit date:
2016-02-02

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