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Interferon-γ couples CD8+ T cell avidity and differentiation during infection

Abstract:
Effective responses to intracellular pathogens are characterized by T cell clones with a broad affinity range for their cognate peptide and diverse functional phenotypes. How T cell clones are selected throughout the response to retain a breadth of avidities remains unclear. Here, we demonstrate that direct sensing of the cytokine IFN-γ by CD8+ T cells coordinates avidity and differentiation during infection. IFN-γ promotes the expansion of low-avidity T cells, allowing them to overcome the selective advantage of high-avidity T cells, whilst reinforcing high-avidity T cell entry into the memory pool, thus reducing the average avidity of the primary response and increasing that of the memory response. IFN-γ in this context is mainly provided by virtual memory T cells, an antigen-inexperienced subset with memory features. Overall, we propose that IFN-γ and virtual memory T cells fulfil a critical immunoregulatory role by enabling the coordination of T cell avidity and fate.
Publication status:
Published
Peer review status:
Peer reviewed

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Publisher copy:
10.1038/s41467-023-42455-4

Authors


More by this author
Institution:
University of Oxford
Division:
MSD
Department:
NDORMS
Sub department:
Kennedy Institute for Rheumatology
Role:
Author
More by this author
Institution:
University of Oxford
Division:
MSD
Department:
NDORMS
Sub department:
Kennedy Institute for Rheumatology
Role:
Author
ORCID:
0000-0002-1148-1650
More by this author
Institution:
University of Oxford
Division:
MSD
Department:
NDORMS
Sub department:
Kennedy Institute for Rheumatology
Role:
Author
More by this author
Institution:
University of Oxford
Division:
MSD
Department:
NDORMS
Sub department:
Kennedy Institute for Rheumatology
Role:
Author
More by this author
Institution:
University of Oxford
Division:
MSD
Department:
NDORMS
Sub department:
Kennedy Institute for Rheumatology
Role:
Author


More from this funder
Funding agency for:
Gerard, A
Grant:
0006162
More from this funder
Funding agency for:
Gerard, A
Grant:
KENN151607
KENN202112
More from this funder
Funding agency for:
Gerard, A
Grant:
C5255/A18085
More from this funder
Funding agency for:
Uhl, LFK


Publisher:
Springer Nature
Journal:
Nature Communications More from this journal
Volume:
14
Article number:
6727
Publication date:
2023-10-23
Acceptance date:
2023-10-11
DOI:
EISSN:
2041-1723


Language:
English
Pubs id:
1552292
Local pid:
pubs:1552292
Deposit date:
2023-10-24

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