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Dynamic mitochondrial transcription and translation in B cells control germinal center entry and lymphomagenesis

Abstract:
Germinal center (GC) B cells undergo proliferation at very high rates in a hypoxic microenvironment but the cellular processes driving this are incompletely understood. Here we show that the mitochondria of GC B cells are highly dynamic, with significantly upregulated transcription and translation rates associated with the activity of transcription factor A, mitochondrial (TFAM). TFAM, while also necessary for normal B cell development, is required for entry of activated GC precursor B cells into the germinal center reaction; deletion of Tfam significantly impairs GC formation, function and output. Loss of TFAM in B cells compromises the actin cytoskeleton and impairs cellular motility of GC B cells in response to chemokine signaling, leading to their spatial disorganization. We show that B cell lymphoma substantially increases mitochondrial translation and that deletion of Tfam in B cells is protective against the development of lymphoma in a c-Myc transgenic mouse model. Finally, we show that pharmacological inhibition of mitochondrial transcription and translation inhibits growth of GC-derived human lymphoma cells and induces similar defects in the actin cytoskeleton.
Publication status:
Published
Peer review status:
Peer reviewed

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Publisher copy:
10.1038/s41590-023-01484-3

Authors




Publisher:
Springer Nature
Journal:
Nature Immunology More from this journal
Volume:
24
Issue:
6
Pages:
991–1006
Publication date:
2023-04-24
Acceptance date:
2023-03-09
DOI:
EISSN:
1529-2916
ISSN:
1529-2908


Language:
English
Keywords:
Pubs id:
1334132
Local pid:
pubs:1334132
Deposit date:
2023-03-24

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