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Increasing radiosensitivity by alleviating tumour hypoxia: a drug screen reveals atovaquone as a clinical candidate

Abstract:
Tumour hypoxia renders cancer cells resistant to cancer therapy, resulting in markedly worse clinical outcomes. To find clinical candidate compounds that reduce hypoxia in tumours, we conducted a high throughput screen for oxygen consumption rate (OCR) reduction and identified a number of drugs with this property. For this study we focused on the antimalarial, atovaquone. Atovaquone rapidly decreases the OCR by more than 80% in a wide range of cancer cell lines at pharmacological concentrations. In addition, atovaquone eradicates hypoxia in FaDu, HCT116 and H1299 spheroids. Similarly, it virtually eliminates hypoxia in FaDu and HCT116 xenografts in nude mice, and causes a significant tumour growth delay when combined with radiation. Atovaquone is a ubiquinone analogue, and decreases the OCR by inhibiting mitochondrial complex III. We are now undertaking clinical studies to assess whether atovaquone reduces tumour hypoxia in patients, thereby increasing the efficacy of radiotherapy.
Publication status:
Published
Peer review status:
Peer reviewed

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Publisher copy:
10.1038/ncomms12308

Authors

More by this author
Institution:
University of Oxford
Division:
MSD
Department:
Oncology
Role:
Author
More by this author
Institution:
University of Oxford
Division:
MSD
Department:
Oncology
Role:
Author
More by this author
Institution:
University of Oxford
Division:
MSD
Department:
Oncology
Role:
Author


Publisher:
Nature Publishing Group
Journal:
Nature Communications More from this journal
Volume:
7
Article number:
12308
Publication date:
2016-06-01
Acceptance date:
2016-06-17
DOI:
ISSN:
2041-1723


Pubs id:
pubs:633477
UUID:
uuid:c0976293-ae5d-4422-95a2-d5cf0077bcd9
Local pid:
pubs:633477
Source identifiers:
633477
Deposit date:
2016-07-12
ARK identifier:

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