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Tead4 and Tfap2c generate bipotency and a bistable switch in totipotent embryos to promote robust lineage diversification

Abstract:
The mouse and human embryo gradually loses totipotency before diversifying into the inner cell mass (ICM, future organism) and trophectoderm (TE, future placenta). The transcription factors TFAP2C and TEAD4 with activated RHOA accelerate embryo polarization. Here we show that these factors also accelerate the loss of totipotency. TFAP2C and TEAD4 paradoxically promote and inhibit Hippo signaling before lineage diversification: they drive expression of multiple Hippo regulators while also promoting apical domain formation, which inactivates Hippo. Each factor activates TE specifiers in bipotent cells, while TFAP2C also activates specifiers of the ICM fate. Asymmetric segregation of the apical domain reconciles the opposing regulation of Hippo signaling into Hippo OFF and the TE fate, or Hippo ON and the ICM fate. We propose that the bistable switch established by TFAP2C and TEAD4 is exploited to trigger robust lineage diversification in the developing embryo.
Publication status:
Published
Peer review status:
Peer reviewed

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Publisher copy:
10.1038/s41594-024-01311-9

Authors

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Role:
Author
ORCID:
0000-0001-6157-8840
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Role:
Author
ORCID:
0009-0000-9224-3795
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Institution:
University of Oxford
Division:
MSD
Department:
Physiology Anatomy & Genetics
Role:
Author


Publisher:
Springer Nature
Journal:
Nature Structural & Molecular Biology More from this journal
Volume:
31
Issue:
6
Pages:
964–976
Place of publication:
United States
Publication date:
2024-05-24
Acceptance date:
2024-04-09
DOI:
EISSN:
1545-9985
ISSN:
1545-9993
Pmid:
38789684


Language:
English
Keywords:
Pubs id:
1999790
Local pid:
pubs:1999790
Deposit date:
2024-05-28
ARK identifier:

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