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Journal article

GSDMB is increased in IBD and regulates epithelial restitution/repair independent of pyroptosis

Abstract:
Gasdermins are a family of structurally related proteins originally described for their role in pyroptosis. Gasdermin B (GSDMB) is currently the least studied, and while its association with genetic susceptibility to chronic mucosal inflammatory disorders is well established, little is known about its functional relevance during active disease states. Herein, we report increased GSDMB in inflammatory bowel disease, with single-cell analysis identifying epithelial specificity to inflamed colonocytes/crypt top colonocytes. Surprisingly, mechanistic experiments and transcriptome profiling reveal lack of inherent GSDMB-dependent pyroptosis in activated epithelial cells and organoids but instead point to increased proliferation and migration during in vitro wound closure, which arrests in GSDMB-deficient cells that display hyper-adhesiveness and enhanced formation of vinculin-based focal adhesions dependent on PDGF-A-mediated FAK phosphorylation. Importantly, carriage of disease-associated GSDMB SNPs confers functional defects, disrupting epithelial restitution/repair, which, altogether, establishes GSDMB as a critical factor for restoration of epithelial barrier function and the resolution of inflammation.
Publication status:
Published
Peer review status:
Peer reviewed

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Publisher copy:
10.1016/j.cell.2021.12.024

Authors

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Role:
Author
ORCID:
0000-0002-7020-5808
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Role:
Author
ORCID:
0000-0002-6536-2868
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Role:
Author
ORCID:
0000-0003-0614-1990


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Funder identifier:
https://ror.org/02nv4he32
Grant:
NIHR-RP-R3-12-026
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Funder identifier:
https://ror.org/0187kwz08
Grant:
NIHR201410
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Funder identifier:
https://ror.org/029chgv08
Grant:
102974/Z/13/Z
102974/Z/13/Z
219523/Z/19/Z
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Funder identifier:
https://ror.org/03x94j517
Grant:
MC_PC_MR/S025952/1
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Funder identifier:
https://ror.org/001aqnf71
Grant:
MR/S036377/1


Publisher:
Cell Press
Journal:
Cell More from this journal
Volume:
185
Issue:
2
Pages:
283-298
Place of publication:
United States
Publication date:
2022-01-11
Acceptance date:
2021-12-16
DOI:
EISSN:
1097-4172
ISSN:
0092-8674
Pmid:
35021065


Language:
English
Pubs id:
1232733
Local pid:
pubs:1232733
Deposit date:
2024-09-13
ARK identifier:

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