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Hepcidin is suppressed by erythropoiesis in hemoglobin E β-thalassemia and β-thalassemia trait.

Abstract:

HbE β-thalassemia is the most common severe thalassemia syndrome across Asia, and millions are carriers. Clinical heterogeneity in HbE β-thalassemia is incompletely explained by genotype, and the interaction of phenotypic variation with hepcidin is unknown. The effect of thalassemia carriage on hepcidin is also unknown, but could be relevant for iron supplementation programs aimed at combating anemia. In 69 Sri Lankan HbE β-thalassemia patients with moderate or severe phenotype, hepcidin was ...

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Journal:
Blood More from this journal
Publication date:
2014-12-01
DOI:
EISSN:
1528-0020
ISSN:
0006-4971
Language:
English
Pubs id:
pubs:501351
UUID:
uuid:a7f692e7-c190-4eeb-bf91-d6b6af39adf1
Local pid:
pubs:501351
Source identifiers:
501351
Deposit date:
2015-01-02

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