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Journal article

Gain-of-function mutations in KCNK3 cause a developmental disorder with sleep apnea

Abstract:
Sleep apnea is a common disorder that represents a global public health burden. KCNK3 encodes TASK-1, a K+ channel implicated in the control of breathing, but its link with sleep apnea remains poorly understood. Here we describe a new developmental disorder with associated sleep apnea (developmental delay with sleep apnea, or DDSA) caused by rare de novo gain-of-function mutations in KCNK3. The mutations cluster around the ‘X-gate’, a gating motif that controls channel opening, and produce overactive channels that no longer respond to inhibition by G-protein-coupled receptor pathways. However, despite their defective X-gating, these mutant channels can still be inhibited by a range of known TASK channel inhibitors. These results not only highlight an important new role for TASK-1 K+ channels and their link with sleep apnea but also identify possible therapeutic strategies.
Publication status:
Published
Peer review status:
Peer reviewed

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Publisher copy:
10.1038/s41588-022-01185-x

Authors


More by this author
Institution:
University of Oxford
Division:
MPLS
Department:
Physics
Role:
Author
ORCID:
0000-0002-3288-6681
More by this author
Role:
Author
ORCID:
0000-0002-6192-5651
More by this author
Institution:
University of Oxford
Division:
MPLS
Department:
Physics
Role:
Author
More by this author
Institution:
University of Oxford
Division:
MPLS
Department:
Physics
Role:
Author
ORCID:
0000-0001-5867-994X
More by this author
Institution:
University of Oxford
Role:
Author
ORCID:
0000-0003-4892-5523


Publisher:
Nature Research
Journal:
Nature Genetics More from this journal
Volume:
54
Issue:
10
Pages:
1534-1543
Publication date:
2022-10-04
Acceptance date:
2022-08-09
DOI:
EISSN:
1546-1718
ISSN:
1061-4036
Pmid:
36195757


Language:
English
Keywords:
Pubs id:
1282109
Local pid:
pubs:1282109
Deposit date:
2022-10-17

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