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PTCH1-mutant human cerebellar organoids exhibit altered neural development and recapitulate early medulloblastoma tumorigenesis

Abstract:
Patched 1 (PTCH1) is the primary receptor for the sonic hedgehog (SHH) ligand and negatively regulates SHH signalling, an essential pathway in human embryogenesis. Loss-of-function mutations in PTCH1 are associated with altered neuronal development and the malignant brain tumour medulloblastoma. As a result of differences between murine and human development, molecular and cellular perturbations that arise from human PTCH1 mutations remain poorly understood. Here, we used cerebellar organoids differentiated from human induced pluripotent stem cells combined with CRISPR/Cas9 gene editing to investigate the earliest molecular and cellular consequences of PTCH1 mutations on human cerebellar development. Our findings demonstrate that developmental mechanisms in cerebellar organoids reflect in vivo processes of regionalisation and SHH signalling, and offer new insights into early pathophysiological events of medulloblastoma tumorigenesis without the use of animal models.
Publication status:
Published
Peer review status:
Peer reviewed

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Publisher copy:
10.1242/dmm.050323

Authors

More by this author
Institution:
University of Oxford
Division:
MSD
Department:
Clinical Neurosciences
Role:
Author
ORCID:
0000-0001-6168-0116
More by this author
Institution:
University of Oxford
Division:
MSD
Department:
Clinical Neurosciences
Role:
Author
ORCID:
0000-0001-5682-0671
More by this author
Institution:
University of Oxford
Division:
MSD
Department:
Pathology Dunn School
Role:
Author
ORCID:
0000-0002-5357-8231
More by this author
Role:
Author
ORCID:
0000-0003-3364-1587
More by this author
Role:
Author
ORCID:
0000-0002-1220-5252


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Funder identifier:
https://ror.org/01k33dw46
Grant:
P01 CA096832
More from this funder
Funder identifier:
https://ror.org/03x94j517
Grant:
MR/V037730/1


Publisher:
Company of Biologists
Journal:
Disease Models & Mechanisms More from this journal
Volume:
17
Issue:
2
Article number:
dmm050323
Place of publication:
England
Publication date:
2024-02-27
Acceptance date:
2024-02-06
DOI:
EISSN:
1754-8411
ISSN:
1754-8403
Pmid:
38411252

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