Journal article icon

Journal article

Abcc5 knockout mice have lower fat mass and increased levels of circulating GLP‐1

Abstract:
Objective A previous genome‐wide association study linked overexpression of an ATP‐binding cassette transporter, ABCC5, in humans with a susceptibility to developing type 2 diabetes with age. Specifically, ABCC5 gene overexpression was shown to be strongly associated with increased visceral fat mass and reduced peripheral insulin sensitivity. Currently, the role of ABCC5 in diabetes and obesity is unknown. This study reports the metabolic phenotyping of a global Abcc5 knockout mouse.

Methods A global Abcc5‐/‐ mouse was generated by CRISPR/Cas9. Fat mass was determined by weekly EchoMRI and fat pads were dissected and weighed at week 18. Glucose homeostasis was ascertained by an oral glucose tolerance test, intraperitoneal glucose tolerance test, and intraperitoneal insulin tolerance test. Energy expenditure and locomotor activity were measured using PhenoMaster cages. Glucagon‐like peptide 1 (GLP‐1) levels in plasma, primary gut cell cultures, and GLUTag cells were determined by enzyme‐linked immunosorbent assay.

Results Abcc5‐/‐ mice had decreased fat mass and increased plasma levels of GLP‐1, and they were more insulin sensitive and more active. Recombinant overexpression of ABCC5 protein in GLUTag cells decreased GLP‐1 release.

Conclusions ABCC5 protein expression levels are inversely related to fat mass and appear to play a role in the regulation of GLP‐1 secretion from enteroendocrine cells.
Publication status:
Published
Peer review status:
Peer reviewed

Actions


Access Document


Files:
Publisher copy:
10.1002/oby.22521

Authors


More by this author
Institution:
University of Oxford
Division:
Medical Sciences Division
Department:
Physiology Anatomy and Genetics
Oxford college:
St Catherine's College
Role:
Author
More by this author
Institution:
University of Oxford
Division:
Medical Sciences Division
Department:
Physiology Anatomy and Genetics
Role:
Author
More by this author
Institution:
University of Oxford
Division:
Medical Sciences Division
Department:
Physiology Anatomy and Genetics
Oxford college:
Lincoln College
Role:
Author
More by this author
Institution:
University of Oxford
Division:
Medical Sciences Division
Department:
Physiology Anatomy and Genetics
Role:
Author
More by this author
Institution:
University of Oxford
Division:
Medical Sciences Division
Department:
Physiology Anatomy and Genetics
Role:
Author


More from this funder
Funding agency for:
Cyranka, M
De Wet, H
Grant:
BB/P020666/1
BB/P020666/1
More from this funder
Funding agency for:
Cyranka, M
De Wet, H
Grant:
BB/P020666/1
BB/P020666/1
More from this funder
Funding agency for:
Cyranka, M
De Wet, H
Grant:
BB/P020666/1
BB/P020666/1
More from this funder
Funding agency for:
Cantley, J
More from this funder
Funding agency for:
Veprik, A


Publisher:
Wiley
Journal:
Obesity More from this journal
Volume:
27
Issue:
8
Pages:
1292-1304
Publication date:
2019-07-24
Acceptance date:
2019-04-09
DOI:
EISSN:
1930-739X
ISSN:
1930-7381
Pmid:
31338999


Language:
English
Pubs id:
pubs:1036789
UUID:
uuid:66a660c8-b712-4a61-afb9-64ba64aa795f
Local pid:
pubs:1036789
Source identifiers:
1036789
Deposit date:
2019-08-02

Terms of use



Views and Downloads






If you are the owner of this record, you can report an update to it here: Report update to this record

TO TOP