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Aldehyde-mediated inhibition of asparagine biosynthesis has implications for diabetes and alcoholism

Abstract:
Patients with alcoholism and type 2 diabetes manifest altered metabolism, including elevated aldehyde levels and unusually low asparagine levels. We show that asparagine synthetase B (ASNS), the only human asparagine-forming enzyme, is inhibited by disease-relevant reactive aldehydes, including formaldehyde and acetaldehyde. Cellular studies show non-cytotoxic amounts of reactive aldehydes induce a decrease in asparagine levels. Biochemical analyses reveal inhibition results from reaction of the aldehydes with the catalytically important N-terminal cysteine of ASNS. The combined cellular and biochemical results suggest a possible mechanism underlying the low asparagine levels in alcoholism and diabetes. The results will stimulate research on the biological consequences of the reactions of aldehydes with nucleophilic residues.
Publication status:
Published
Peer review status:
Peer reviewed

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Publisher copy:
10.1039/d3sc06551k

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Role:
Author
ORCID:
0000-0002-6123-595X
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Role:
Author
ORCID:
0000-0002-1287-9580
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Role:
Author
ORCID:
0000-0002-6297-2986



Publisher:
Royal Society of Chemistry
Journal:
Chemical Science More from this journal
Volume:
15
Issue:
7
Pages:
2509-2517
Publication date:
2024-01-15
Acceptance date:
2024-01-01
DOI:
EISSN:
2041-6539
ISSN:
2041-6520
Pmid:
38362406


Language:
English
Keywords:
Pubs id:
1607236
Local pid:
pubs:1607236
Deposit date:
2024-05-01

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