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Journal article

Hyperglycaemia induces metabolic dysfunction and glycogen accumulation in pancreatic β-cells

Abstract:
Insulin secretion from pancreatic β-cells is impaired in all forms of diabetes. The resultant hyperglycaemia has deleterious effects on many tissues, including β-cells. Here we use a mouse model of human neonatal diabetes to show that chronic hyperglycemia impairs glucose metabolism and alters expression of metabolic genes in pancreatic islets. This results in marked glycogen accumulation, and increased apoptosis in β-cells. Sulphonylurea therapy rapidly normalizes blood glucose levels, dissipates glycogen stores, increases autophagy, and restores β-cell metabolism. Insulin therapy has the same effect but with slower kinetics. Similar changes are observed in mice expressing an activating glucokinase mutation, in in vitro models of hyperglycaemia, and in islets from type-2 diabetes patients. Altered β-cell metabolism may underlie both the progressive impairment of insulin secretion and reduced β-cell mass in diabetes.
Publication status:
Published
Peer review status:
Peer reviewed

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Publisher copy:
10.1038/ncomms13496

Authors

More by this author
Institution:
University of Oxford
Division:
MSD
Department:
Physiology Anatomy & Genetics
Role:
Author
More by this author
Institution:
University of Oxford
Division:
MSD
Department:
Physiology Anatomy & Genetics
Role:
Author
More by this author
Institution:
University of Oxford
Division:
MSD
Department:
Physiology Anatomy & Genetics
Role:
Author


More from this funder
Funding agency for:
Ashcroft, F
Grant:
Research Wolfson Merit Award
More from this funder
Funding agency for:
Rohm, M
Grant:
postdoctoral fellowship
More from this funder
Funding agency for:
Ashcroft, F
Grant:
Research Wolfson Merit Award
Advanced grant 322620
More from this funder
Funding agency for:
Rohm, M
Grant:
postdoctoral fellowship
More from this funder
Funding agency for:
Brereton, M
Grant:
OXION Training Fellowship
884655, 089795


Publisher:
Nature Publishing Group
Journal:
Nature Communications More from this journal
Volume:
7
Article number:
13496
Publication date:
2016-11-24
Acceptance date:
2016-10-07
DOI:
ISSN:
2041-1723


Keywords:
Pubs id:
pubs:648543
UUID:
uuid:44f29080-48ef-42d9-a457-80bbd134cb77
Local pid:
pubs:648543
Deposit date:
2016-10-10
ARK identifier:

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