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Development of macrocycle kinase inhibitors for ALK2 using Fibrodysplasia ossificans progressiva‐derived endothelial cells

Abstract:

Fibrodysplasia ossificans progressiva (FOP) is an extremely rare congenital form of heterotopic ossification (HO), caused by heterozygous mutations in the Activin A type I receptor (ACVR1), that encodes the bone morphogenetic protein (BMP) type I receptor ALK2. These mutations enable ALK2 to induce downstream signaling in response to Activins, thereby turning them into bone inducing agents. To date there is no cure for FOP. The further development of FOP patient‐derived models may contribute to discover novel biomarkers and therapeutic approaches. Nevertheless, this has traditionally been a challenge, as biopsy sampling often triggers HO.
We have characterized peripheral blood‐derived endothelial colony forming cells (ECFCs) from three independent FOP donors as a new model for FOP. FOP ECFCs are prone to undergo Endothelial‐to‐mesenchymal transition and exhibit increased ALK2 downstream signaling and subsequent osteogenic differentiation upon stimulation with Activin A. Moreover, we have identified a new class of small molecule macrocycles with potential activity against ALK2 kinase. Finally, using FOP ECFCs, we have selected OD36 and OD52 as potent inhibitors with excellent kinase selectivity profiles that potently antagonize mutant ALK2 signaling and osteogenic differentiation. We expect that these results will contribute to the development of novel ALK2 clinical candidates for the treatment of FOP.

Publication status:
Published
Peer review status:
Peer reviewed

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Publisher copy:
10.1002/jbm4.10230

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More by this author
Institution:
University of Oxford
Division:
MSD
Department:
NDM
Sub department:
Structural Genomics Consortium
Role:
Author
ORCID:
0000-0001-6757-0436
et al.



Publisher:
Wiley
Journal:
JBMR Plus More from this journal
Volume:
3
Issue:
11
Pages:
e10230
Publication date:
2019-10-07
Acceptance date:
2019-08-06
DOI:
EISSN:
2473-4039


Keywords:
Pubs id:
pubs:1039497
UUID:
uuid:3f89f36a-ce63-4508-940b-02ce522b0264
Local pid:
pubs:1039497
Source identifiers:
1039497
Deposit date:
2019-08-07

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