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Development of macrocycle kinase inhibitors for ALK2 using Fibrodysplasia ossificans progressiva‐derived endothelial cells

Abstract:

Fibrodysplasia ossificans progressiva (FOP) is an extremely rare congenital form of heterotopic ossification (HO), caused by heterozygous mutations in the Activin A type I receptor (ACVR1), that encodes the bone morphogenetic protein (BMP) type I receptor ALK2. These mutations enable ALK2 to induce downstream signaling in response to Activins, thereby turning them into bone inducing agents. To date there is no cure for FOP. The further development of FOP patie...

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Publication status:
Published
Peer review status:
Peer reviewed

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Publisher copy:
10.1002/jbm4.10230

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Institution:
University of Oxford
Division:
MSD
Department:
NDM
Sub department:
Structural Genomics Consortium
Role:
Author
ORCID:
0000-0001-6757-0436
et al.
Publisher:
Wiley Publisher's website
Journal:
JBMR Plus Journal website
Volume:
3
Issue:
11
Pages:
e10230
Publication date:
2019-10-07
Acceptance date:
2019-08-06
DOI:
EISSN:
2473-4039
Keywords:
Pubs id:
pubs:1039497
UUID:
uuid:3f89f36a-ce63-4508-940b-02ce522b0264
Local pid:
pubs:1039497
Source identifiers:
1039497
Deposit date:
2019-08-07

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