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Parkin-mediated ubiquitination contributes to the constitutive turnover of mitochondrial fission factor (Mff)

Abstract:

The mitochondrial outer membrane protein Mitochondrial Fission Factor (Mff) plays a key role in both physiological and pathological fission. It is well established that at stressed or functionally impaired mitochondria, PINK1 recruits the ubiquitin ligase Parkin which ubiquitinates Mff and other mitochondrial outer membrane proteins to facilitate the removal of defective mitochondria and maintain the integrity of the mitochondrial network. Here we show that, in addition to this clearance path...

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Publication status:
Published
Peer review status:
Peer reviewed

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Publisher copy:
10.1371/journal.pone.0213116

Authors


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Institution:
University of Oxford
Division:
MSD
Department:
NDORMS
Role:
Author
ORCID:
0000-0002-7974-6803
More by this author
Role:
Author
ORCID:
0000-0002-8904-5795
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Role:
Author
ORCID:
0000-0002-0660-4126
More by this author
Role:
Author
ORCID:
0000-0002-8115-8592
More by this author
Role:
Author
ORCID:
0000-0003-3589-8335
British Heart Foundation More from this funder
Publisher:
Public Library of Science Publisher's website
Journal:
PloS One Journal website
Volume:
14
Issue:
5
Article number:
e0213116
Place of publication:
United States
Publication date:
2019-05-21
Acceptance date:
2019-05-06
DOI:
EISSN:
1932-6203
ISSN:
1932-6203
Pmid:
31112535

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