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Guanidinoacetate N-methyltransferase-knockout mice completely lacking myocardial phosphocreatine show normal in vivo cardiac function, but increased susceptibility to ischemia/reperfusion injury

Publication status:
Published

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Institution:
University of Oxford
Department:
Oxford, MSD, RDM, Cardiovascular Medicine, BHF Centre of Research Excellence
Role:
Author
More by this author
Institution:
University of Oxford
Department:
Oxford, MSD, RDM, Cardiovascular Medicine, BHF Centre of Research Excellence
Role:
Author
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Volume:
110
Issue:
17
Pages:
203-203
Publication date:
2004-10-26
ISSN:
0009-7322
URN:
uuid:344ab1ed-a16a-449a-b8e9-d9e6530c59ba
Source identifiers:
104264
Local pid:
pubs:104264

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