Journal article
Conveying the pathogenesis of type 1 diabetes to the blind, low‐vision and diverse needs communities through sensory stimulation
- Abstract:
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To educate members of the blind, low-vision and diverse needs communities on the pathogenesis of the chronic autoimmune disease, type 1 diabetes, members of our team with research expertise in immune-mediated diseases, participated in the 2023 Monash Sensory Science (MSS) Exhibition. Using QR code linked audio commentary, participants were guided through tactile displays demonstrating normal insulin action in the regulation of blood glucose levels and its vital role in providing energy to tissues, followed by displays describing the various stages of the immune system's aberrant attack and the eventual complete destruction of the insulin producing beta-cells of the pancreatic islets in type 1 diabetes. These models conveyed to the participants the huge effect that this autoimmune-mediated disease has on the quality of life of affected individuals including the subsequent lifelong reliance on insulin injections to maintain glucose homeostasis. This MSS Exhibition provided a unique opportunity for our researchers to engage with under-represented members of the community and to raise awareness about such a debilitating and common autoimmune disease.
- Publication status:
- Published
- Peer review status:
- Peer reviewed
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- Files:
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(Preview, Version of record, pdf, 860.2KB, Terms of use)
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- Publisher copy:
- 10.1111/imcb.12741
Authors
- Publisher:
- Wiley
- Journal:
- Immunology and Cell Biology More from this journal
- Volume:
- 102
- Issue:
- 5
- Pages:
- 341-346
- Publication date:
- 2024-03-05
- DOI:
- EISSN:
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1440-1711
- ISSN:
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0818-9641
- Language:
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English
- Keywords:
- Pubs id:
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2080257
- Local pid:
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pubs:2080257
- Deposit date:
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2025-01-23
Terms of use
- Copyright holder:
- Tran et al.
- Copyright date:
- 2024
- Rights statement:
- © 2024 The Authors. Immunology & Cell Biology published by John Wiley & Sons Australia, Ltd on behalf of the Australian and New Zealand Society for Immunology, Inc. This is an open access article under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.
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