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Macrophage anti-inflammatory behaviour in a multiphase model of atherosclerotic plaque development

Abstract:
Atherosclerosis is an inflammatory disease characterised by the formation of plaques, which are deposits of lipids and cholesterol-laden macrophages that form in the artery wall. The inflammation is often non-resolving, due in large part to changes in normal macrophage anti-inflammatory behaviour that are induced by the toxic plaque microenvironment. These changes include higher death rates, defective efferocytic uptake of dead cells, and reduced rates of emigration. We develop a free boundary multiphase model for early atherosclerotic plaques, and we use it to investigate the effects of impaired macrophage anti-inflammatory behaviour on plaque structure and growth. We find that high rates of cell death relative to efferocytic uptake results in a plaque populated mostly by dead cells. We also find that emigration can potentially slow or halt plaque growth by allowing material to exit the plaque, but this is contingent on the availability of live macrophage foam cells in the deep plaque. Finally, we introduce an additional bead species to model macrophage tagging via microspheres, and we use the extended model to explore how high rates of cell death and low rates of efferocytosis and emigration prevent the clearance of macrophages from the plaque.
Publication status:
Published
Peer review status:
Peer reviewed

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Publisher copy:
10.1007/s11538-023-01142-7

Authors

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Role:
Author
ORCID:
0000-0002-1745-2770
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Institution:
University of Oxford
Division:
MPLS
Department:
Mathematical Institute
Oxford college:
Keble College
Role:
Author
ORCID:
0000-0003-1771-5910


Publisher:
Springer
Journal:
Bulletin of Mathematical Biology More from this journal
Volume:
85
Issue:
5
Article number:
37
Publication date:
2023-03-29
Acceptance date:
2023-03-09
DOI:
EISSN:
1522-9602
ISSN:
0092-8240


Language:
English
Keywords:
Pubs id:
1336352
Local pid:
pubs:1336352
Deposit date:
2023-04-28
ARK identifier:

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