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Journal article

Alcohol consumption and telomere length: Mendelian randomization clarifies alcohol’s effects

Abstract:
Alcohol’s impact on telomere length, a proposed marker of biological aging, is unclear. We performed the largest observational study to date (in n = 245,354 UK Biobank participants) and compared findings with Mendelian randomization (MR) estimates. Two-sample MR used data from 472,174 participants in a recent genome-wide association study (GWAS) of telomere length. Genetic variants were selected on the basis of associations with alcohol consumption (n = 941,280) and alcohol use disorder (AUD) (n = 57,564 cases). Non-linear MR employed UK Biobank individual data. MR analyses suggested a causal relationship between alcohol traits, more strongly for AUD, and telomere length. Higher genetically-predicted AUD (inverse variance-weighted (IVW) β = −0.06, 95% confidence interval (CI): −0.10 to −0.02, p = 0.001) was associated with shorter telomere length. There was a weaker association with genetically-predicted alcoholic drinks weekly (IVW β = −0.07, CI: −0.14 to −0.01, p = 0.03). Results were consistent across methods and independent from smoking. Non-linear analyses indicated a potential threshold relationship between alcohol and telomere length. Our findings indicate that alcohol consumption may shorten telomere length. There are implications for age-related diseases.
Publication status:
Published
Peer review status:
Peer reviewed

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Publisher copy:
10.1038/s41380-022-01690-9

Authors


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Institution:
University of Oxford
Division:
MSD
Department:
Nuffield Department of Population Health
Role:
Author
ORCID:
0000-0002-8408-0372


Publisher:
Springer Nature
Journal:
Molecular Psychiatry More from this journal
Volume:
27
Pages:
4001-4008
Publication date:
2022-07-26
Acceptance date:
2022-06-29
DOI:
EISSN:
1476-5578
ISSN:
1359-4184


Language:
English
Keywords:
Pubs id:
1265052
Local pid:
pubs:1265052
Deposit date:
2022-06-23

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