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Trans-ethnic kidney function association study reveals putative causal genes and effects on kidney-specific disease aetiologies

Abstract:
Chronic kidney disease (CKD) affects ~10% of the global population, with considerable ethnic differences in prevalence and aetiology. We assemble genome-wide association studies of estimated glomerular filtration rate (eGFR), a measure of kidney function that defines CKD, in 312,468 individuals of diverse ancestry. We identify 127 distinct association signals with homogeneous effects on eGFR across ancestries and enrichment in genomic annotations including kidney-specific histone modifications. Fine-mapping reveals 40 high-confidence variants driving eGFR associations and highlights putative causal genes with cell-type specific expression in glomerulus, and in proximal and distal nephron. Mendelian randomisation supports causal effects of eGFR on overall and cause-specific CKD, kidney stone formation, diastolic blood pressure and hypertension. These results define novel molecular mechanisms and putative causal genes for eGFR, offering insight into clinical outcomes and routes to CKD treatment development.
Publication status:
Published
Peer review status:
Peer reviewed

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Publisher copy:
10.1038/s41467-018-07867-7

Authors

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Institution:
University of Oxford
Division:
Medical Sciences Division
Department:
NDM
Sub department:
Human Genetics Wt Centre
Role:
Author
ORCID:
0000-0002-6805-6014
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Role:
Author
ORCID:
0000-0001-8450-3518


Publisher:
Springer Nature
Journal:
Nature Communications More from this journal
Volume:
10
Article number:
29
Publication date:
2019-01-03
Acceptance date:
2018-12-03
DOI:
ISSN:
2041-1723
Pmid:
30604766


Language:
English
Pubs id:
pubs:958252
UUID:
uuid:325f32f7-3aa0-41fb-8885-9a7fc47142ad
Local pid:
pubs:958252
Source identifiers:
958252
Deposit date:
2019-01-16
ARK identifier:

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