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Changes in amylin and amylin-like peptide concentrations and beta-cell function in response to sulfonylurea or insulin therapy in NIDDM.

Abstract:

OBJECTIVE: Amylin, a secretory peptide of beta-cells, is the constituent peptide of islet amyloid, which is characteristic of NIDDM, and changes in amylin secretion in response to therapies may influence the rate of production of islet amyloid. The primary objective of this study was to determine whether therapy with sulfonylurea or basal insulin in NIDDM would alter amylin secretion in a way that might affect the formation of islet amyloid. RESEARCH DESIGN AND METHODS: In a randomized cross...

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Publication status:
Published

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Publisher copy:
10.2337/diacare.21.5.810

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Institution:
University of Oxford
Department:
Oxford, MSD, RDM, OCDEM
Role:
Author
More by this author
Institution:
University of Oxford
Department:
Oxford, MSD, RDM, OCDEM, Biomedical Research Centre
Role:
Author
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Journal:
Diabetes care
Volume:
21
Issue:
5
Pages:
810-816
Publication date:
1998-05-05
DOI:
EISSN:
1935-5548
ISSN:
0149-5992
URN:
uuid:ff39d087-8dcc-4926-a2b9-cc184a2a2b85
Source identifiers:
2716
Local pid:
pubs:2716

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