- Abstract:
-
OBJECTIVE: Amylin, a secretory peptide of beta-cells, is the constituent peptide of islet amyloid, which is characteristic of NIDDM, and changes in amylin secretion in response to therapies may influence the rate of production of islet amyloid. The primary objective of this study was to determine whether therapy with sulfonylurea or basal insulin in NIDDM would alter amylin secretion in a way that might affect the formation of islet amyloid. RESEARCH DESIGN AND METHODS: In a randomized cross...
Expand abstract - Publication status:
- Published
- Publisher copy:
- 10.2337/diacare.21.5.810
-
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- Journal:
- Diabetes care
- Volume:
- 21
- Issue:
- 5
- Pages:
- 810-816
- Publication date:
- 1998-05-05
- DOI:
- EISSN:
-
1935-5548
- ISSN:
-
0149-5992
- URN:
-
uuid:ff39d087-8dcc-4926-a2b9-cc184a2a2b85
- Source identifiers:
-
2716
- Local pid:
- pubs:2716
- Language:
- English
- Keywords:
- Copyright date:
- 1998
Journal article
Changes in amylin and amylin-like peptide concentrations and beta-cell function in response to sulfonylurea or insulin therapy in NIDDM.
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