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Soluble complement receptor 1 (sCR1) protects against experimental autoimmune myasthenia gravis

Abstract:: The loss of muscle function seen in myasthenia gravis and in the animal model of the disease, experimental autoimmune myasthenia gravis (EAMG) is in part due to the activation of complement by anti-acetylcholine receptor (AChR) antibodies at the motor end-plate. In this study we describe the effects of a soluble recombinant form of human complement receptor 1 (sCR1) on the development of clinical disease and receptor loss in EAMG induced passively by administr...
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Publication status:: Published

Peer review status:: Peer reviewed

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APA Style

Piddlesden, S., Jiang, S., Levin, J., Vincent, A., & Morgan, B. (1997). Soluble complement receptor 1 (sCR1) protects against experimental autoimmune myasthenia gravis. Journal of Neuroimmunology, 71(1-2), 173–177.

MLA Style

Piddlesden, S., et al. “Soluble Complement Receptor 1 (sCR1) Protects against Experimental Autoimmune Myasthenia Gravis.” Journal of Neuroimmunology, vol. 71, no. 1-2, Elsevier, 1997, pp. 173–77.

Chicago Style

Piddlesden, S, S Jiang, J Levin, A Vincent, and B Morgan. 1997. “Soluble Complement Receptor 1 (sCR1) Protects against Experimental Autoimmune Myasthenia Gravis.” Journal of Neuroimmunology 71 (1-2): 173–77.
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Publisher copy:: 10.1016/s0165-5728(96)00144-0

Authors

+ Piddlesden, S More by this author

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Author

+ Jiang, S More by this author

Institution:

University of Oxford

Division:

MSD

Department:

Oncology

Role:

Author

+ Levin, J More by this author

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Author

+ Vincent, A More by this author

Role:

Author

+ Morgan, B More by this author

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Author

Bibliographic Details

Publisher:: Elsevier Publisher's website
Journal:: Journal of Neuroimmunology Journal website
Volume:: 71
Issue:: 1-2
Pages:: 173-177
Publication date:: 1997-11-07
Acceptance date:: 1996-07-31
DOI:: 10.1016/s0165-5728(96)00144-0
ISSN:: 0165-5728

Item Description

Language:: English
Keywords:: SBTMR
Pubs id:: pubs:1025961
UUID:: uuid:cce49dea-7730-43d7-94f3-8c7061a5ad9e
Local pid:: pubs:1025961
Source identifiers:: 1025961
Deposit date:: 2019-07-02

Terms of use

Copyright holder:: Elsevier
Copyright date:: 1997
Notes:: Copyright © 1996 Elsevier Science B.V. All rights reserved.

Licence:: Terms and Conditions of Use for Oxford University Research Archive

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