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Journal article

Myasthenic syndromes due to defects in COL13A1 and in the N‐linked glycosylation pathway

Abstract:

The congenital myasthenic syndromes (CMS) are hereditary disorders of neuromuscular transmission. The number of cases recognized, at around 1:100,000 in the United Kingdom, is increasing with improved diagnosis. The advent of next‐generation sequencing has facilitated the discovery of many genes that harbor CMS‐associated mutations. An emerging group of CMS, characterized by a limb‐girdle pattern of muscle weakness, is caused by mutations in genes that encode proteins involved in the initial ...

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Publication status:
Published
Peer review status:
Peer reviewed

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Publisher copy:
10.1111/nyas.13576

Authors


More by this author
Institution:
University of Oxford
Division:
MSD
Department:
Clinical Neurosciences
Oxford college:
St Cross College
Role:
Author
More by this author
Institution:
University of Oxford
Division:
MSD
Department:
Clinical Neurosciences
Role:
Author
More by this author
Institution:
University of Oxford
Division:
MSD
Department:
Clinical Neurosciences
Role:
Author
More by this author
Institution:
University of Oxford
Division:
MSD
Department:
Clinical Neurosciences
Role:
Author
More by this author
Institution:
University of Oxford
Division:
MSD
Department:
Clinical Neurosciences
Role:
Author
More from this funder
Grant:
MR/M006824/1
MR/M006824/1
Publisher:
Wiley Publisher's website
Journal:
Annals of the New York Academy of Sciences Journal website
Volume:
1413
Issue:
1
Pages:
163-169
Place of publication:
United States
Publication date:
2018-01-24
Acceptance date:
2017-11-14
DOI:
EISSN:
1749-6632
ISSN:
0077-8923
Pmid:
29363764
Language:
English
Keywords:
Pubs id:
821316
Local pid:
pubs:821316
Deposit date:
2020-02-17

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