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Arginine transport and other determinants of nitric oxide production in human septic shock

Abstract:

The arterial vasodilation seen in human septic shock is conventionally attributed to increased nitric oxide (NO) production, primarily by extrapolation of animal and cellular studies. Little is known of the cellular source of NO in human septic shock. Other mediators, such as carbon monoxide (CO), may modulate NO production, and could also directly contribute to vasodilation.

This study has examined the NO and CO synthetic pathways in peripheral blood mononuclear cells and mesenter...

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Institution:
University of Oxford
Department:
Medical Sciences Division
Role:
Author

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Role:
Supervisor
Role:
Supervisor
Publication date:
2002
Type of award:
DPhil
Level of award:
Doctoral
Awarding institution:
University of Oxford
Barcode:
603824078
URN:
uuid:abf881fc-3c9a-40b4-89e1-07b8df2067d1
Local pid:
td:603824078
Language:
English
Subjects:

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