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Modulation of the NO pathway during short or prolonged blood reperfusion following ischaemia in a heterotopic rat heart transplantation model.

Abstract:

Ischemia-reperfusion injury plays a major role in graft dysfunction following transplantation. Extensive research has demonstrated that nitric oxide (NO) plays a fundamental role to protect the heart against this injury. Consequently, we quantified NO synthase (NOS) isoform protein levels in a rat heart transplant model during short and prolonged reperfusion following ischemia. Experiments were performed using a modified Lewis to Lewis heterotopic abdominal heart transplantation with a total ...

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Publication status:
Published

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Authors


Desrois, M More by this author
Durrans, A More by this author
More by this author
Institution:
University of Oxford
Department:
Oxford, MSD, Physiology Anatomy and Genetics, BHF Centre of Research Excellence
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Journal:
Transplantation proceedings
Volume:
36
Issue:
5
Pages:
1280-1282
Publication date:
2004-06-05
DOI:
EISSN:
1873-2623
ISSN:
0041-1345
URN:
uuid:a68869f5-7690-429f-8441-9186f5404691
Source identifiers:
106732
Local pid:
pubs:106732

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