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Mitochondrial dysfunction and mitophagy in Parkinson's: from familial to sporadic disease.

Abstract:

Parkinson's disease (PD) is a progressive neurodegenerative disorder characterised by the preferential loss of dopaminergic neurons in the substantia nigra. Mitochondrial dysfunction is increasingly appreciated as a key determinant of dopaminergic neuronal susceptibility in PD and is a feature of both familial and sporadic disease, as well as in toxin-induced Parkinsonism. Recently, the mechanisms by which PD-associated mitochondrial proteins phosphatase and tensin homolog deleted on chromoso...

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Publication status:
Published
Peer review status:
Peer reviewed

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Publisher copy:
10.1016/j.tibs.2015.02.003

Authors


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Institution:
University of Oxford
Division:
MSD
Department:
Physiology Anatomy & Genetics
Role:
Author
More by this author
Institution:
University of Oxford
Division:
MSD
Department:
Physiology Anatomy & Genetics
Role:
Author
More by this author
Institution:
University of Oxford
Division:
MSD
Department:
Physiology Anatomy & Genetics
Role:
Author
More from this funder
Grant:
G-0801
Monument Discovery Award
G-1003
Publisher:
Elsevier
Journal:
Trends in Biochemical Sciences More from this journal
Volume:
40
Issue:
4
Pages:
200-210
Publication date:
2015-03-08
DOI:
EISSN:
1362-4326
ISSN:
0968-0004
Language:
English
Keywords:
Pubs id:
pubs:510096
UUID:
uuid:a2dbf60e-828b-4f62-b5a2-036776e2882c
Local pid:
pubs:510096
Source identifiers:
510096
Deposit date:
2016-01-28

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