- Abstract:
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Developmental dyslexia is a common disorder with a strong genetic component, but the underlying molecular mechanisms are still unknown. Several candidate dyslexia susceptibility genes, including KIAA0319, DYX1C1 and DCDC2, have been identified in humans. RNA interference experiments targeting these genes in rat embryos have shown impairments in neuronal migration, suggesting that defects in radial cortical migration could be involved in the disease mechanism of dyslexia. Here we present the f...
Expand abstract - Publication status:
- Published
- Peer review status:
- Peer reviewed
- Version:
- Accepted manuscript
- Files:
-
-
(pdf, 97.6MB)
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- Publisher copy:
- 10.1007/s00429-016-1282-1
-
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- Publisher:
- Springer Publisher's website
- Journal:
- Brain Structure & Function Journal website
- Publication date:
- 2016-08-05
- Acceptance date:
- 2016-07-26
- DOI:
- ISSN:
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1863-2661 and 1863-2653
- Pubs id:
-
pubs:638527
- URN:
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uri:a0d7451c-555c-487f-a748-3c33af76a7a5
- UUID:
-
uuid:a0d7451c-555c-487f-a748-3c33af76a7a5
- Local pid:
- pubs:638527
- Language:
- English
- Keywords:
- Copyright holder:
- Velayos-Baeza et al
- Copyright date:
- 2016
- Notes:
- © The Author(s) 2016 This article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made.
Journal article
Normal radial migration and lamination are maintained in dyslexia-susceptibility candidate gene homolog Kiaa0319 knockout mice.
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