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Tenascin-C modulates matrix contraction via focal adhesion kinase- and Rho-mediated signaling pathways.

Abstract:

A provisional matrix consisting of fibrin and fibronectin (FN) is deposited at sites of tissue damage and repair. This matrix serves as a scaffold for fibroblast migration into the wound where these cells deposit new matrix to replace lost or damaged tissue and eventually contract the matrix to bring the margins of the wound together. Tenascin-C is expressed transiently during wound repair in tissue adjacent to areas of injury and contacts the provisional matrix in vivo. Using a synthetic mod...

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Publisher copy:
10.1091/mbc.e02-05-0292

Authors


Journal:
Molecular biology of the cell
Volume:
13
Issue:
10
Pages:
3601-3613
Publication date:
2002-10-05
DOI:
EISSN:
1939-4586
ISSN:
1059-1524
URN:
uuid:8f8a6bd7-f81f-4a6a-b6ff-d8f7f50d99a6
Source identifiers:
225493
Local pid:
pubs:225493

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