-308 tumor necrosis factor (TNF) polymorphism is not associated with survival in severe sepsis and is unrelated to lipopolysaccharide inducibility of the human TNF promoter.

Stuber, F; Udalova, IA; Book, M; Drutskaya, LN; Kuprash, DV; Turetskaya, RL; Schade, FU; Nedospasov, SA

Abstract:

Tumor necrosis factor (TNF) is recognized as a central mediator of sepsis, septic shock, and multiple organ failure. These host reactions are associated with increased TNF levels in circulation, presumably due to increased TNF production. A previously described nucleotide variation at position -308 in the promoter region of the human TNF gene was shown to be associated with the clinical outcome of malaria. In this study we addressed the relevance of the -308 polymorphism for expression of the...

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APA Style

Stuber, F., Udalova, I. A., Book, M., Drutskaya, L. N., Kuprash, D. V., Turetskaya, R. L., Schade, F. U., & Nedospasov, S. A. (1995). -308 tumor necrosis factor (TNF) polymorphism is not associated with survival in severe sepsis and is unrelated to lipopolysaccharide inducibility of the human TNF promoter. Journal of Inflammation, 46(1), 42–50.

MLA Style

Stuber, F., et al. “-308 Tumor Necrosis Factor (TNF) Polymorphism Is Not Associated with Survival in Severe Sepsis and Is Unrelated to Lipopolysaccharide Inducibility of the Human TNF Promoter.” Journal of Inflammation, vol. 46, no. 1, 1995, pp. 42–50.

Chicago Style

Stuber, F, IA Udalova, M Book, LN Drutskaya, DV Kuprash, RL Turetskaya, FU Schade, and SA Nedospasov. 1995. “-308 Tumor Necrosis Factor (TNF) Polymorphism Is Not Associated with Survival in Severe Sepsis and Is Unrelated to Lipopolysaccharide Inducibility of the Human TNF Promoter.” Journal of Inflammation 46 (1): 42–50.
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