- Abstract:
- In a recent study, Leucci et al. report a role for the long non-coding RNA SAMMSON in driving mitochondrial function in melanoma. Targeting SAMMSON, the gene of which is frequently co-amplified with MITF, highlights a new cell-type-specific therapeutic vulnerability in melanoma irrespective of BRAF, NRAS, or p53 status.
- Publication status:
- Published
- Peer review status:
- Peer reviewed
- Version:
- Accepted Manuscript
- Files:
-
-
(pdf, 110.5kb)
-
- Publisher copy:
- 10.1016/j.ccell.2016.04.010
-
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- Publisher:
- Cell Press Publisher's website
- Journal:
- Cancer Cell Journal website
- Volume:
- 29
- Issue:
- 5
- Pages:
- 619–621
- Publication date:
- 2016
- DOI:
- EISSN:
-
1878-3686
- ISSN:
-
1535-6108
- URN:
-
uuid:6c97790c-51b6-4653-a285-a1ff95285a8d
- Source identifiers:
-
619465
- Local pid:
- pubs:619465
- Copyright holder:
- Elsevier Inc.
- Copyright date:
- 2016
- Notes:
-
Copyright © 2016 Elsevier Inc. This is the accepted manuscript version of the article. The final version is available online from Elsevier at: https://doi.org/10.1016/j.ccell.2016.04.010
Journal article
Targeting the lncRNA SAMMSON reveals metabolic vulnerability in melanoma
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