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Subthalamic nucleus stimulation–induced local field potential changes in dystonia

Abstract:

Background
Subthalamic nucleus (STN) stimulation is an effective treatment for Parkinson's disease and induced local field potential (LFP) changes that have been linked with clinical improvement. STN stimulation has also been used in dystonia although the internal globus pallidus is the standard target where theta power has been suggested as a physiomarker for adaptive stimulation.

Objective
We aimed to explore if enhanced theta power was also present in STN and if stimulation-induced spectral changes that were previously reported for Parkinson's disease would occur in dystonia.

Methods
We recorded LFPs from 7 patients (12 hemispheres) with isolated craniocervical dystonia whose electrodes were placed such that inferior, middle, and superior contacts covered STN, zona incerta, and thalamus.

Results
We did not observe prominent theta power in STN at rest. STN stimulation induced similar spectral changes in dystonia as in Parkinson's disease, such as broadband power suppression, evoked resonant neural activity (ERNA), finely-tuned gamma oscillations, and an increase in aperiodic exponents in STN-LFPs. Both power suppression and ERNA localize to STN. Based on this, single-pulse STN stimulation elicits evoked neural activities with largest amplitudes in STN, which are relayed to the zona incerta and thalamus with changing characteristics as the distance from STN increases.

Conclusions
Our results show that STN stimulation–induced spectral changes are a nondisease-specific response to high-frequency stimulation, which can serve as placement markers for STN. This broadens the scope of STN stimulation and makes it an option for other disorders with excessive oscillatory peaks in STN. © 2022 The Authors. Movement Disorders published by Wiley Periodicals LLC on behalf of International Parkinson and Movement Disorder Society.

Publication status:
Published
Peer review status:
Peer reviewed

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Publisher copy:
10.1002/mds.29302

Authors


More by this author
Institution:
University of Oxford
Division:
MSD
Department:
Clinical Neurosciences
Oxford college:
Wolfson College
Role:
Author
ORCID:
0000-0001-5728-3951
More by this author
Role:
Author
ORCID:
0000-0002-9834-3639
More by this author
Institution:
University of Oxford
Role:
Author
ORCID:
0000-0003-4484-9949
More by this author
Institution:
University of Oxford
Role:
Author
More by this author
Institution:
University of Oxford
Role:
Author


Publisher:
Wiley
Journal:
Movement Disorders More from this journal
Volume:
38
Issue:
3
Pages:
423-434
Publication date:
2022-12-23
Acceptance date:
2022-12-05
DOI:
EISSN:
1531-8257
ISSN:
0885-3185


Language:
English
Keywords:
Pubs id:
1317080
Local pid:
pubs:1317080
Deposit date:
2022-12-24

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