Beer, N. L., Tribble, N. D., Colclough, K., Arundel, P., Grimsby, J., Chik, C., Ellard, S., & Gloyn, A. L. (2010). Naturally occurring glucokinase mutations at the same amino acid residue cause opposite clinical phenotypes of hypo- and hyperglycaemia. 53, S202–S202.

Beer, N. L., et al. Naturally Occurring Glucokinase Mutations at the Same Amino Acid Residue Cause Opposite Clinical Phenotypes of Hypo- and Hyperglycaemia. 2010, pp. S202–S202.

Beer, NL, ND Tribble, K Colclough, P Arundel, J Grimsby, C Chik, S Ellard, and AL Gloyn. 2010. “Naturally Occurring Glucokinase Mutations at the Same Amino Acid Residue Cause Opposite Clinical Phenotypes of Hypo- and Hyperglycaemia.” In , 53:S202–S202.