Beer, N., Tribble, N., Colclough, K., Arundel, P., Grimsby, J., Chik, C., Ellard, S., & Gloyn, A. (2016). Naturally occurring glucokinase mutations at the same amino acid residue cause opposite clinical phenotypes of hypo- and hyperglycaemia. 53, S202–S202.

Beer, N., et al. Naturally Occurring Glucokinase Mutations at the Same Amino Acid Residue Cause Opposite Clinical Phenotypes of Hypo- and Hyperglycaemia. 2016, pp. S202–S202.

Beer, N, N Tribble, K Colclough, P Arundel, J Grimsby, C Chik, S Ellard, and A Gloyn. 2016. “Naturally Occurring Glucokinase Mutations at the Same Amino Acid Residue Cause Opposite Clinical Phenotypes of Hypo- and Hyperglycaemia.” In , 53:S202–S202.