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Airway epithelial cells generate pro-inflammatory tenascin-C and small extracellular vesicles in response to TLR3 stimuli and rhinovirus infection

Abstract:

Viral infections are a common cause of asthma exacerbations, with human rhinoviruses (RV) the most common trigger. RV signals through a number of different receptors, including toll-like receptor (TLR)3. Tenascin-C (TN-C) is an immunomodulatory extracellular matrix protein present in high quantities in the airway of people with asthma, and expression is also upregulated in nasal lavage fluid in response to RV infection. Respiratory viral infection has been demonstrated to induce the release o...

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Publication status:
Published
Peer review status:
Peer reviewed

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Publisher copy:
10.3389/fimmu.2019.01987

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Institution:
University of Oxford
Division:
Medical Sciences Division
Department:
NDORMS
Role:
Author
Publisher:
Frontiers Media Publisher's website
Journal:
Frontiers in Immunology
Volume:
10
Article number:
1987
Publication date:
2019-08-21
Acceptance date:
2019-08-06
DOI:
EISSN:
1664-3224
Pmid:
31497021
Language:
English
Keywords:
Pubs id:
pubs:1049124
UUID:
uuid:12afd546-b55f-4102-b089-8b718aeacad5
Local pid:
pubs:1049124
Source identifiers:
1049124
Deposit date:
2019-09-30

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