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Genetic inhibition of galectin-3 suppressed cardiac fibrosis and improved function in a mouse model of dilated cardiomyopathy

Abstract:
The adult human heart has very little capacity to repair damage because adult cardiomyocytes lack the ability to proliferate; the immune system plays a very important role in modulating this response. The immune system of fetuses possesses diminished pro-inflammatory function, and an immature immune system, which is believed to provide an immunological setting in which cardiomyocytes can regenerate.
Publication status:
Published
Peer review status:
Peer reviewed

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Publisher copy:
10.1016/j.hlc.2017.06.210

Authors


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Institution:
University of Oxford
Division:
MSD
Department:
RDM
Sub department:
RDM Cardiovascular Medicine
Role:
Author
Publisher:
Elsevier
Host title:
65th Annual Scientific Meeting of the Cardiac Society of Australia and New Zealand
Journal:
65th Annual Scientific Meeting of the Cardiac Society of Australia and New Zealand More from this journal
Publication date:
2017-08-02
Acceptance date:
2017-05-11
DOI:
ISSN:
1443-9506
Pubs id:
pubs:856647
UUID:
uuid:09c40acc-7f7d-4753-81df-0b243338a532
Local pid:
pubs:856647
Source identifiers:
856647
Deposit date:
2018-06-08

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